Killing Me Unsoftly
نویسندگان
چکیده
منابع مشابه
Killing Me Unsoftly
The aorta, as the major conduit artery of the human body, is primarily thought of as a blood vessel that provides a low-resistance path for blood flow directed from the heart to peripheral organs and tissues. However, the aorta has another central hemodynamic function, whereby the elastic nature of the aortic wall provides a significant biomechanical buffering capacity complementing the pulsati...
متن کاملKilling me softly: myth in pharmaceutical advertising.
Although the influence of research onmedical practice has become a key concern, the influence of pharmaceutical advertising in medical journals has received little attention. There is evidence that advertising influences doctors’ behaviour more than they might think. Important pockets of research exist in this area but tend to focus on the scientific validity of the text and rarely give much at...
متن کاملKilling Me Softly: The Fetal Origins Hypothesis.
In the epidemiological literature, the fetal origins hypothesis associated with David J. Barker posits that chronic, degenerative conditions of adult health, including heart disease and type 2 diabetes, may be triggered by circumstance decades earlier, in utero nutrition in particular. Economists have expanded on this hypothesis, investigating a broader range of fetal shocks and circumstances a...
متن کاملKilling Me Softly—Future Challenges in Apoptosis Research
The induction of apoptosis, a highly regulated and clearly defined mode of cell dying, is a vital tenet of modern cancer therapy. In this review we focus on three aspects of apoptosis research which we believe are the most crucial and most exciting areas currently investigated and that will need to be better understood in order to enhance the efficacy of therapeutic measures. First, we discuss ...
متن کاملKilling Me Softly: Connotations to Unfolded Protein Response and Oxidative Stress in Alzheimer's Disease
This review is focused on the possible causes of mitochondrial dysfunction in AD, underlying molecular mechanisms of this malfunction, possible causes and known consequences of APP, Aβ, and hyperphosphorylated tau presence in mitochondria, and the contribution of altered lipid metabolism (nonsterol isoprenoids) to pathological processes leading to increased formation and accumulation of the afo...
متن کاملذخیره در منابع من
با ذخیره ی این منبع در منابع من، دسترسی به آن را برای استفاده های بعدی آسان تر کنید
ژورنال
عنوان ژورنال: Arteriosclerosis, Thrombosis, and Vascular Biology
سال: 2017
ISSN: 1079-5642,1524-4636
DOI: 10.1161/atvbaha.116.308563